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The long-term goal of the Henrickson Lab is to understand the role of T cell dysfunction in chronic allergic and inflammatory disease, including asthma and obesity, as well as rare, monogenic primary immunodeficiency. To this end, the lab uses complementary approaches in humans and mice, leveraging detailed immunophenotyping and metabolic characterization of immune dysfunction in patients to identify mechanistic hypotheses to test in mouse models and human cells in vitro.
Sarah E. Henrickson, MD, PhD
Dr. Henrickson investigates the mechanisms of T cell dysfunction in monogenic primary immunodeficiency and chronic inflammatory disease, including asthma and obesity, and primary immunodeficiency.